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From: TSS ()
Subject: Re: SEAC ANNUAL REPORT 2006
Date: February 24, 2007 at 7:41 pm PST

In Reply to: SEAC ANNUAL REPORT 2006 posted by TSS on February 2, 2007 at 8:25 am:

----- Original Message -----
From: Terry S. Singeltary Sr.
To: XXXXXXXX XXXXXXX (SEAC)
Sent: Friday, February 23, 2007 9:49 AM
Subject: Re: SEAC ANNUAL REPORT 2006

THANK YOU very much for your kind reply.
I look forward to seeing this report, and hope that it is not of a 'spontaneous' nature. ...

with kindest regards,
I am sincerely,

Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518

----- Original Message -----
From: XXXXXXXX XXXXXXXXXX (SEAC)
To: 'flounder9@verizon.net'
Sent: Friday, February 23, 2007 8:13 AM
Subject: RE: SEAC ANNUAL REPORT 2006

Dear Mr Singeltary

Thank you for your email about a possible relationship between BASE and sCJD.

SEAC is continually informed about and considers the emerging science on both animal and human TSEs. The committee regularly receives updates from the UK's National CJD Surveillance Centre Unit on the epidemiology of vCJD and sCJD and is also aware of emerging research on BASE. It is envisaged that SEAC will conduct a detailed consideration of the available science and the possible animal and human health implications of BASE at its meeting on 10th May 2007.

Yours sincerely

XXXXXXXXX XXXXXXXXXXXX

----------------------------------------------------------
From: Terry S. Singeltary Sr. [mailto:flounder9@verizon.net]
Sent: 02 February 2007 16:44
To: Bovine Spongiform Encephalopathy
Cc: SEACsecretariat (AHEG); cjdvoice@yahoogroups.com; BLOODCJD@YAHOOGROUPS.COM; madcow@lists.iatp.org; Sustainable Agriculture Network Discussion Group
Subject: SEAC ANNUAL REPORT 2006

Date: February 2, 2007 at 8:25 am PST
SEAC ANNUAL REPORT 2006 (ignoring the obvious)

http://www.seac.gov.uk/publicats/annualreport2006.pdf

i guess the sporadic CJD cases have all be resolved, and the route and cause classified.
to this day, i cannot figure this out. if BSE farmers died from sporadic CJD, and now another study seems to point
that BSE and BASE may be the same thing, and that BASE does not produce vCJD like pathology, but pathology resembling that of sporadic CJD, then why does SEAC still seem to put sporadic CJD on the back burners ??? IN fact, SEAC could not even speak about it in there 2006 report. would it not seem prudent to mention these findings in this 2006 SEAC report ;

Other work presented suggested that BSE and bovine amyloidotic spongiform
encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the
prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A
MUTATION FOUND IN CASES OF SPORADIC CJD.

snip...

http://www.seac.gov.uk/minutes/95.pdf

***These results indicate that BASE is transmissible to humans and suggest that BASE is more virulent than
classical BSE in humans.***

6:30 Close of Day One

http://www.healthtech.com/2007/tse/day1.asp

64. A member noted that at the recent Neuroprion meeting, a study was
presented showing that in transgenic mice BSE passaged in sheep may be more
virulent and infectious to a wider range of species than bovine derived BSE.

http://www.seac.gov.uk/minutes/95.pdf

Asante/Collinge et al, that BSE transmission to the 129-methionine

genotype can lead to an alternate phenotype that is indistinguishable

from type 2 PrPSc, the commonest _sporadic_ CJD;

http://www.fda.gov/ohrms/dockets/ac/03/slides/3923s1_OPH.htm

I STILL think to ignore these findings below is not only rediculous, but indeed very suspicious ;

CJD FARMERS WIFE 1989

http://www.bseinquiry.gov.uk/files/yb/1989/10/13007001.pdf

http://www.bseinquiry.gov.uk/files/yb/1989/10/13003001.pdf

cover-up of 4th farm worker ???

http://www.bseinquiry.gov.uk/files/yb/1995/10/23006001.pdf

http://www.bseinquiry.gov.uk/files/yb/1995/10/20006001.pdf

CONFIRMATION OF CJD IN FOURTH FARMER

http://www.bseinquiry.gov.uk/files/yb/1995/11/03008001.pdf

now story changes from;

SEAC concluded that, if the fourth case were confirmed, it would be
worrying, especially as all four farmers with CJD would have had BSE
cases on their farms.

to;

This is not unexpected...

was another farmer expected?

http://www.bseinquiry.gov.uk/files/yb/1995/11/13010001.pdf

4th farmer, and 1st teenager

http://www.bseinquiry.gov.uk/files/yb/1996/02/27003001.pdf

2. snip...

Over a 5 year period, which is the time period on which the advice
from Professor Smith and Dr. Gore was based, and assuming a
population of 120,000 dairy farm workers, and an annual incidence
of 1 per million cases of CJD in the general population, a
DAIRY FARM WORKER IS 5 TIMES MORE LIKELY THAN
an individual in the general population to develop CJD. Using the
actual current annual incidence of CJD in the UK of 0.7 per
million, this figure becomes 7.5 TIMES.

3. You will recall that the advice provided by Professor Smith in
1993 and by Dr. Gore this month used the sub-population of dairy
farm workers who had had a case of BSE on their farms -
63,000, which is approximately half the number of dairy farm
workers - as a denominator. If the above sums are repeated using
this denominator population, taking an annual incidence in the general
population of 1 per million the observed rate in this sub-population
is 10 TIMES, and taking an annual incidence of 0.7 per million,
IT IS 15 TIMES (THE ''WORST CASE'' SCENARIO) than
that in the general population...

http://www.bseinquiry.gov.uk/files/yb/1995/01/31004001.pdf

CJD YOUNG PEOPLE

in the USA, a 16 year old in 1978;

ALSO IN USA;

(20 year old died from sCJD in USA in 1980 and a 16 year
old in 1981. see second url below)

in France, a 19 year old in 1982;

in Canada, a 14 year old of UK origin in 1988;

in Poland, cases in people aged 19, 23, and 27 were identified in
a retrospective study (published 1991), having been originally
misdiagnosed with a viral encephalitis;

Creutzfeldt's first patient in 1923 was aged 23.

http://www.bseinquiry.gov.uk/files/yb/1995/10/27013001.pdf

20 year old died from sCJD in USA in 1980 and a 16 year
old in 1981. A 19 year old died from sCJD in
France in 1985. There is no evidence of an iatrogenic
cause for those cases....

http://www.bseinquiry.gov.uk/files/yb/1995/10/04004001.pdf

FOR SEAC TO continue to flounder with this ukbsenvcjd only theory, when we know that sporadic CJD's were proven long ago to transmit via the medical and surgical arena, will only continue to spread this agent around the globe via a multitude of proven routes and sources.

IN my opinion, for SEAC to continue to go down this same road, year after year, and continue to ignore sporadic CJD's, and continue to say that "BSE in the UK sheep population is most likely to be zero, or very low if present at all." "Consequently, any impact of the schemes on human health from removing BSE from sheep is likely to be negligible." when in fact they do not know. and in fact science is showing that in "transgenic mice BSE passaged in sheep may be more virulent and infectious to a wider range of species than bovine derived BSE." IN my opinion this continued neglect of other TSEs in human and animals, the continued belief in this ukbsenvcjd only theory, and the risk thereof, is reckless, dangerous, and in my mind, criminal. SEAC has failed the public terribly in this regard. ...TSS

SNIP.....END...........TSS




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